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We hypothesized that the confined action of these Wnt agonists and antagonists on matrix cells is predominantly achieved by restricting the expression of the molecular machinery, required to mediate the activity of Rspondins and Dkk2, to matrix cells

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Currently, it is documented that the major redox control for these enzymes is a feedback inhibition exerted by GSH on GCL (Meyer and Fricker, 2002)

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