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Through complex mechanisms, antiretroviral therapies and HIV virulence factors reportedly increase ROS that consume GSH while HIV-1 Tat, a protein necessary for efficient transcription, seems to downregulate enzymes in de novo synthesis [57,58]

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: Vasoactive intestinal peptide stimulation of adenylate cyclase and active electrolyte secretion in intestinal mucosa

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Given the oxidative stress caused by -CG administration 7 , we proposed that ROS play a critical role in elevating autophagy activity in enterocytes

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