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and Streptococcus mutans on hydrophobicity, hemolysin production and biofilms and their synergy with antibiotics

Conclusion: Normal brain GSH in living brain of chronic MA users is consistent with our previous postmortem brain finding and suggests that any oxidative stress caused by MA, at the doses used by our participants, might not be sufficient to cause either a compensatory increase in, or substantial overutilization of, this antioxidant

The cells were washed with PBS and harvested

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