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SCFAs activate GPCRs (GPR43/41) and inhibit histone deacetylases (HDAC), promoting regulatory T-cell (Treg) differentiation while suppressing Th17 cell polarization within the joint, thereby restoring immune homeostasis and reducing pro-inflammatory cytokine release
Further transcriptome evaluation followed by RT-qPCR analysis allowed us to pin down several DEHP-induced functional enzymes involved in the metabolic process
The bioregulators were applied for the first 2-3 years of observation
