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[DOI] [PubMed] [Google Scholar] 322.Shen Q

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They employed mechanism-based affinity labeling to modify the putative active site of GGT using 2-amino-4-(fluorophosphono) butanoic acid (1), a -phosphonic acid monofluoride derivative of glutamic acid

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Intrinsic (mitochondrial) apoptosis is initiated by mitochondrial damage, leading to outer membrane permeabilization and cytochrome c release