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650 Ferroptosis in traumatic brain injury Traumatic brain injury (TBI) is a significant global cause of disability and mortality, 651 with complex underlying mechanisms such as excitatory neurotoxicity, apoptosis, oxidative stress, inflammatory response, and intracellular calcium overload

however, the coexistence of both forms is possible

Elevated ROS and IL-10 recruit the transcription of matrix metalloproteinases (MMPs) which recruit neuro-vasculature and remodel the ECM, such that we have endometriotic tissue adhering to the structures of the pelvic bowl

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Aldo-keto reductases mediate constitutive and inducible protection against aldehyde toxicity in human neuroblastoma SH-SY5Y cells