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The results shown by the present study strongly suggest that NAC administration inhibited ethanol-induced oxidative stress (GSSG/GSH ratio) activating the Nrf2-ARE system, which is in line with a report that an analog of NAC, NACA, attenuated the oxidative stress induced by a traumatic brain injury in rats via the same antioxidant system (Zhou et al., Whether an activation of xCT transporter function due to an increased substrate availability and/or an increased xCT expression plays a role in the NAC-induced reduction of hippocampal GSSG/GSH ratio, the administration of the xCT inhibitor SZ was expected to reduce or block such an effect of NAC

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After reconstitution with BAC water, refrigerate at 28C and use within 812 weeks

Mention of a commercial or proprietary product does not constitute an endorsement or recommendation for its use by the USDA

Cisplatin: mode of cytotoxic action and molecular basis of resistance

Front Pharmacol, 2017