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The rapid rate of this half reaction is partially attributed to the low p K a (approximately 3.5) of the N-terminal thiol 11

BACH1 equilibrates the levels of globin and heme through repressing globin and HO1 in erythroblasts upon iron lacking, Accurately adapting erythroblasts to iron deficiency and preventing toxic accumulation of nonheme globin are dependent on the ironhemeBACH1 axis

Understanding these differences is essential for informed decision-making and safe clinical practice

Mar;52(3):74959

[DOI] [PubMed] [Google Scholar] Khafagy el S, Morishita M, Isowa K, Imai J, Takayama K

Mitochondrial diseases: the contribution of organelle stress responses to pathology