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Perikamana SKM, Lee J, Kim EM, Byun H, Lee S, Shin H
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These results suggest that GLR3.3 plays a dominant role in regulating GSH-induced [Ca 2+ ] cyt propagation

Increased IR expression and glycolysis restriction led to the functional and metabolic exhaustion of diabetogenic T cells independent of PD-1 and LAG-3 signaling, as checkpoint blockade failed to reverse this phenotype ( Figure 7 )

Burley CEMK-9 overexpresses a SAT and OASTL gene, whereas Burley TRI-2 harbors extra copies of SAT , GSH1 , and PCS genes
