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Furthermore, the fact that altering NADPH production either in the mitochondria or in the cytosol has the same effects on the GSSG/GSH ratio and on ER stress points to mechanisms for transmission of NADP(H) and/or glutathione status across membranes

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Abstract:Chronic hyperglycemia in diabetics causes microvascular damage through four mechanisms of biochemical changes, including activated protein kinase C (PKC) pathway, activated hexosamine pathway, increased polyol pathway, and increased advanced glycation end-products (AGEs), all of which will increase Reactive Oxygen Species (ROS) levels

pH 8.0), and resuspended in 200 L of elution buffer (10 mM Tris-HCl, 300 mM NaCl, 5 mM EDTA, and 0.5% SDS