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Prior studies [105,106,107] concluded that Cur enhances insulin resistance and glucose homeostasis in mice model of T2DM via the modulation of lipid metabolism

The fixed cells were lysed in an SDS buffer, and the chromatin was fragmented by nuclease digestion

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Conversely, loss of HDAC3 increased ARE activation activity from these sites and diminished cell cycle activity, suggesting that HDAC3 inhibitors may hold promise as a CaP treatment to restore an AR cistrome with more benign features [66]