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This study elucidated the toxic effects and mechanisms of two pollutants in subject animals by validating key genes and metabolites

21 (43%) had been taking acetaminophen for therapeutic purposes

The results suggested that plasma Se 95 g/l represented a functional Se deficit with suboptimal immune status and a deficit in viral handling

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Inflammatory damage to the vascular endothelium, which can be partly mediated by excessive iNOS-derived NO, also results in decreased vascular bioavailability of eNOS-derived NO due to impaired eNOS function [[138], [139], [140]]

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