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The sufficient concentration oxidation of polyunsaturated fatty acids (PUFAs) and phospholipids, the dysregulation of iron metabolism, and the loss of antioxidant defense system execute the ferroptosis [11] and the mechanism of ferroptosis involves a complicated interplay between multiple cellular pathways, including iron metabolism, lipid metabolism, and antioxidant defense mechanisms [12]

It is possible that glycine stimulates the expression of genes for AGAT and GMAT in the skeletal muscle of fish, but, to the best of our knowledge, such experimental data are not available for any animal species

Lv et al., 2015

Research-grade peptides from questionable sources may be cheaper but carry risks

The Gu group first revealed that p53 promotes ferroptosis via a novel tumor suppression mechanism that is independent of canonical p53 functions in cell-cycle arrest and senescence [61, 62]

Delta-tocotrienol (T) treatment could similarly suppress the activity of SLC1A5 by derivatizing glutamate and glutathione, as well as some essential amino acids