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Direct evidence for activity-dependent glucose phosphorylation in neurons with implications for the astrocyte-to-neuron lactate shuttle

This research was supported by grants from the National Institutes of Health and the Office of Dietary Supplements

Endothelial dysfunction, a pathophysiological condition wherein the endothelial homeostasis is disrupted, enhances the risk of thrombosis, inflammation, angiospasm, and intraplaque hemorrhage, resulting in atherothrombosis, infraction, and ischemia [1], and contributes to cardio-metabolic diseases, such as atherosclerosis, acute coronary syndromes, hypertension, reproductive disorders, and diabetes [5,6]

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Support for growth and societal integration is essential