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Alternatively, the activation of receptor tyrosine kinase can inactivate GSK-3 via phosphorylation of upstream kinase Akt or extracellular signal regulated kinase (ERK), thereby allowing cytosolic -catenin to escape ubiquitin-dependent degradation [26]

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(2013) and include correction of particles with a radius less than 0.2 m (Gong, 2003) and dependence on sea surface temperature (Jaegl et al., 2011)

also designed and synthesized a transformable peptide with the ability to self-assemble into micelles in aqueous solution and transform into nanofibrils inhibiting HER2 dimerization and subsequent apoptosis of cancer cells [62]