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While there are established pharmacological guidelines for the prevention of migraines (2, 3), a significant number of patients still experience unmet needs, with many not benefiting from or failing to respond to preventative treatments (4, 5)

Its five main mechanisms of action are as follows: (1) Increases the threshold for migraine attacks by activating different sites on the gamma-aminobutyric acid (GABA) receptors in brain neural tissue, decreasing neuronal excitability, boosting membrane hyperpolarization, and decreasing focal discharges

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DJ-1 acts in parallel to the PINK1/parkin pathway to control mitochondrial function and autophagy

Research indicates that GHK-Cu may affect transcriptional activity associated with matrix synthesis and degradation