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Description
Oxidative stress activates protein tyrosine kinase and matrix metalloproteinases leading to bloodbrain barrier dysfunction
dNP2 is a bloodbrain barrier-permeable peptide enabling ctCTLA-4 protein delivery to ameliorate experimental autoimmune encephalomyelitis
Temporary treatment breaks allow assessment of whether symptoms resolve, helping establish causality
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GSH as a physiological antioxidant fundamentally involved in the maintenance of cellular redox homeostasis 5
