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So, maybe taurine is like the NAD-booster debacle

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This direct transport of extracellular cystine does not take place in hepatocytes, where the reduction of cystine to cysteine occurs mainly in the outer cell membrane as a consequence of GSH efflux (11)

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The expression and activation of YAP and YY1 in the endothelium is triggered by shear stress alterations, and both TFs exert pro-atherogenic effects in vivo, which [43, 44] could be further enhanced by lactylation

NADPH plays a crucial role in the synthesis of various biomolecules, such as fatty acids and amino acids, and is also essential for maintaining the cells antioxidant defenses by donating electrons to reduce oxidized glutathione