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Cell 135 , 907918 (2008)

Integration of proteomics, lipidomics, and metabolomics reveals novel metabolic mechanisms underlying N, N-dimethylformamide induced hepatotoxicity

Blum et al (2003) presented a new family of bioisostere inhibitors, based on the structure of AG 538, a tyrphostin inhibiting the IGF-1R TK at the substrate level and not at the ATP binding site (Blum et al, 2000)

All data were then anonymized

The association between biofilm formation and antimicrobial resistance with possible ingenious bio-remedial approaches

Are Trigger Point Injections Safe
