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Collectively, these observations establish that NRF2 alone can sustain sufficient transcriptional output to maintain cancer cell line growth and that metabolic reprogramming of cells driven by NRF2 does not require redox buffering via enhanced glutathione production

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10.1016/j.biortech.2011.11.103 [DOI] [PubMed] [Google Scholar] Yusof, Y

Multi-dimensional analysis indicated that the proposed system has good stability and biological safety

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