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These data demonstrate that GGCT enzymatically diverts Gln flux away from the TCA cycle, preferentially shunting Gln toward GSH synthesis to sustain redox homeostasis

10.1074/jbc.M113.462341 171 Kamgar-DayhoffP.BrelidzeT

Alleviation of the macrophage cholesterol burden is critical to the inhibition of plaque progression as excess membrane FC promotes inflammatory signaling and cell death (Discussed later)

10.1016/j.chom.2023.11.021 Cell Host Microbe 93 MeguidN

We discovered a glutathione regulator in the ER that likely plays a key role in these conditions. Discovery of a Protein-Folding Regulator in the ER The team determined that this regulator acts as a proofreader, helping ensure that proteins formed in the ER are folded correctly