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Molecular mechanisms regulating NPPCs in IDD Under conditions of elevated oxidative stress, excessive reactive oxygen species (ROS) accumulation and mitochondrial damage activate senescence pathways like P53/P21, contributing to accelerated ECM catabolism and NPPC senescence [76]

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Peer review discussions suggest potential relevance for endothelial and microvascular dysfunction

S., Robinson, E

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For longer-term changes tied to inflammation, focus on strength training, protein and fiber intake, steady sleep, and stress management rather than cleanses or detoxes