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Eventually, the sequential activation of caspase 3 (CASP3), a principal executor of apoptosis, is induced by the activation of initiator CASP8 or CASP10 via cell death receptor pathways, and CASP9 via mitochondrial pathways
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Potential cellular targets and antibacterial efficacy of atmospheric pressure non-thermal plasma
Heterogeneity in the intracellular distribution of the mitochondrial genome further complicates editing efforts [336]
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