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Where These Numbers Come From It's worth being explicit about provenance, because AHK-Cu attracts confident-sounding dose claims that the evidence doesn't support

Together, our observations that a small molecule NNMT inhibitor can increase NAD + and SAM concentrations in differentiated adipocytes and reduce adiposity in DIO mice, suggest that treatment with an NNMT inhibitor effectively reverses obesity via modulation of the NAD + salvage and SAM-mediated (e.g., polyamine flux) pathways to regulate energy expenditure in the adipose tissue

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Glucose Metabolism: Growth hormone is known to impair glucose tolerance and insulin sensitivity, sometimes precipitating diabetes or worsening glycemic control in diabetic patients

The effect on truly "stubborn" subcutaneous fat (which is resistant due to higher alpha-2 adrenergic receptor density) is unclear