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Concluding Remarks Although several mechanisms and cell death pathways are assumed to contribute to the neuronal death in PD, researchers have not conclusively established which might be the primary and most significant mechanism, or if there is a multifactorial cascade for PD pathogenesis

Lu X-Y, Gregory SB, Huda A, Stanley JW

BPC-157 has been studied for its potential interaction with nitric oxide synthase activity and vascular endothelial growth factor (VEGF) signaling, which regulate tissue organization and endothelial function through gene expression modulation rather than direct receptor activation

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Cell Stem Cell 13 , 720733 (2013)