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Interestingly, the GltT-defective mutant also exhibited less adhesive and invasive properties to human bronchial epithelial cells, and transcriptome analysis suggested that genetic inactivation of gltT led to remodeling of the outer membrane and surface structures (Li et al., 2009)

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Footnotes Edited by: Bernd Rosenkranz, Fundisa African Academy of Medicines Development, South Africa Reviewed by: Kacper Nijakowski, Poznan University of Medical Sciences, Poland Miriam Njoki Karinja, Science for Africa Foundation, Kenya Data availability statement The original contributions presented in the study are included in the article/Supplementary Material, further inquiries can be directed to the corresponding author

Clinical, immunological, and molecular findings of patients with p47 phox defect Chronic Granulomatous Disease (CGD) in Indian families

Journal of insect physiology 89 , 18, (2016)