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77,78,79 Genome-wide association studies (GWAS) and genome-wide meta-analyses have identified numerous genetic risk loci associated with SAD, implicating pathways in immune response, lipid metabolism, A plaque, NFTs, and endocytosis, yet many loci remain undiscovered

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in addition to anti-psychotic medication) group in comparison to the placebo group over a 24-week period, but not positive symptoms [7]

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