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Activation of the pro-inflammatory NLRP3 inflammasome/IL-1 axis induces plaque progression and thrombosis through monocyte recruitment, endothelial cell activation and angiogenesis (148, 149)

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Mechanisms and functional implications of intestinal barrier defects

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It also activates focal adhesion kinase (FAK) and paxillin signalling, facilitating cell migration to wound sites

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IL-6 stimulation of insulin-like growth factor binding protein (IGFBP)-1 production

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Headache or Dizziness: Rare and often reversible after changing or stopping use

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After hypoxia, mitochondrial suspensions were isolated from the brains of these rats using the same technique as described above for polarographic measurements [2]

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