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Given the challenge of the undruggable properties of Rho GTPases and the intricate downstream signaling pathways, current strategies for Cdc42 inhibition primarily focus on obstructing interactions with guanine nucleotide exchange factors (GEFs) or inhibiting nucleotide binding [45]

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Despite the widespread use of levothyroxine replacement therapy, a substantial proportion of patients continue to experience persistent symptoms and metabolic dysregulation even after achieving biochemical euthyroidism

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Cadmium and lead interactive effects on oxidative stress and antioxidative responses in rice seedlings