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Coronavirus infection reduces nicotinamide adenine dinucleotide (NAD + ), a cofactor involved in the oxidationreduction reaction, disrupting mitochondrial activity and deacetylase activity of SIRT (48)

Interestingly, recent studies have shown that MAVS plays an important role in regulating human stem cell senescence by maintaining mitochondrial structural and functional homeostasis, independent of the innate immune response [233]

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