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Making memories of stressful events: a journey along epigenetic, gene transcription, and signaling pathways
Enzyme activities (GR, DHAR, APX) were elevated under As stress and further enhanced by treatments, with APX activity increasing by 27% under BRs
Selenium treatment in animal models noted above reduces hyperalgesia, oxidative stress levels, and apoptosis by modulating these channels [138]
consists two distinct genes coding for different SOD such as FeSOD-1 and FeSOD-2 [40]
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HSPs can significantly alter the physiological stress response and promote tolerance [40, 41]
