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TP53, encoded by the TP53 gene and mutated in over 50% of human cancers, is essential for cell-cycle regulation and the induction of programmed cell death

after treatment with 1 mM GSH, there was a 1.5-fold increase in GSH levels and a twofold increase in those of hGSH (Supplementary Table 6)

13C NMR analysis of the cysteine-sulfenic acid redox center of enterococcal NADH peroxidase

Clearly, no single cell type/subtype or unique pathway accounts for all anti-cancer or pro-oncogenic effects of CR in normal, but complex mucosa tissue

Furthermore, no single event occurred in more than one participant
Selenium and vitamin E as antioxidants in chronic hemolytic Anemia: are they deficient
