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Extrinsically, dietary long-chain fatty acids (LCFAs) promote Th17 cells and exacerbate the pathogenesis of experimental autoimmune encephalomyelitis (EAE) ( Figure 1 )

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As the deficiency of the cobalamin-intrinsic factor receptord affects the absorption of vitamin B12 from the ileum into the blood, patients respond well to parenteral vitamin B12 therapy, but not to oral vitamin B12 supplementation (3, 4)

This product is supplied strictly for in-vitro laboratory research use only

A control group was inoculated with PBS while the infection groups received 5x10 (8 )CFU of MAP mutants or wild-type strains ( Figure 1B )