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l glutathione and fatty liver α-Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1α/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

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Unlike somatic cells, which possess a variety of DNA repair pathways to correct oxidative damage, spermatozoa have only one active DNA repair enzyme, 8-oxoguanine DNA glycosylase (OGG1), which is responsible for repairing oxidative base lesions (73, 74)

l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

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l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

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l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

doi: 10.1152/ajpcell.00379.2005 199 SonnevilleF.RuffinM.CorauxC.RousseletN.RouzicP

l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

With a particle size of approximately 160 nm, Bo-TSA-NPs significantly increased epithelial cell uptake via vesicle/caveolae-mediated endocytosis and micropinocytosis

l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

Within thirty minutes, entire signaling pathways activate

l glutathione and fatty liver -Ketoglutarate prevents hyperlipidemia-induced mitochondrial dysfunction oxidative stress by activating the AMPK-pgc-1/Nrf2 pathway Glutathione disulfide sensitizes hepatocytes to

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