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IP GHK-Cu induces sex-dependent activation of mitochondrial, growth, and stress-response pathways rather than the metabolic suppression observed with longer-term IN treatment GSEA was performed to assess transcriptional pathway changes in male and female hippocampi following short-term IP GHK-Cu treatment

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Neurons pretreated with Dihexa demonstrate significantly enhanced survival when exposed to reactive oxygen species, excitotoxic glutamate concentrations, or metabolic impairment compared to untreated controls