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[DOI] [PMC free article] [PubMed] [Google Scholar] 73.Donovan A., Lima C.A., Pinkus J.L., Pinkus G.S., Zon L.I., Robine S., Andrews N.C

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Although the structure of the CBSCH004 complex has not yet been reported, Q222A mutation largely abolishes the inhibitory activity of CH004, suggesting that Gln222 is a key residue for its binding