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Modulation of Transforming Growth Factor-Beta (TGF-Beta) Signaling by Endogenous Sphingolipid Mediators

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Studies have shown that TGF-1 suppresses GCL expression via the JNKATF3 pathway, leading to GSH depletion and enhanced oxidative stress through increased protein and lipid peroxidation [121]

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On matrix remodeling they diverge: BPC-157 acts specifically on tendon fibroblast signaling, TB-500 acts transversally across all repairing tissues via actin dynamics