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Burgenstock, Switzerland, June 26-28

Genetic interactions in the pathogenesis of neonatal hyperbilirubinemia: Gilbert's syndrome and glucose-6-phosphate dehydrogenase deficiency

Ca 2+ homeostasis is essential for optimal oxidative phosphorylation ( 2+ into L-type Ca 2+ channels, which promotes iron accumulation into cardiomyocytes, causing greater oxidative stress (Zhao et al., 2023)

(Minggang Zhu) and M.Z

While the initial success of these agents is encouraging, ferroptosis inhibition may be a double-edged sword in treating radiation injury
