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(2020) presented convincing evidence that elevated TMAO in CVDs could result from the disease-related imbalance in gut microbiota but not the cause of CVDs (Huc et al., 2018)
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We found that 24 h of pre-treatment with ALCAR (1 and 10 mM), resulted in inhibited adhesion of PC-3, DU-145, LNCaP and BPH cells on a fibronectin layer, in a concentration-dependent manner (Fig
