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Figure 3 3.4 Temporal coordination of innate and adaptive immune compartments in gut-associated lymphoid tissue over the course of demyelination and remyelination Given the observed alterations in GM composition, microbial metabolites, and mucus dynamics, we next investigated whether these changes were accompanied by shifts in intestinal immune compartments, focusing on colonic macrophage polarization and T cell populations within gut-associated lymphoid tissue (GALT) ( Figure 4 )

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Likewise, in addition to the mentioned Parkinsons disease models [198,199,200], these were induced by various central causes (e.g., spinal cord compression [170,230], stroke [145], traumatic brain injury [231], neurotoxin cuprizone that mimics multiple sclerosis in rats [232], neuroleptics [199,200], amphetamine [200,233], alcohol acute and chronic intoxication [234], serotonin syndrome [235], and NO-system blockade [200])

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