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J Clin Invest 1985;75:1144-1152

The toxic effects of cisplatin also included triggering an inflammatory response in the cochlea and increased the production of pro-inflammatory cytokines TNF-, IL-1, IL-6, and NF-B (Wang et al., 2004)

Abnormal autophagy and endoplasmic reticulum (ER) damage, which compromise RGC viability due to intracellular accumulation of misfolded proteins, loss of calcium homeostasis and deficient protein trafficking (73) represent additional early features of pressure-related RGC stress, whereas axonal constriction constitutes both a possible early insult and potential coup de grce that instigates apoptosis

Journal of Arrhythmia , 33 , 220225

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