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Until well-designed human clinical trials specifically examine BPC-157 for neuropathy, the compound remains investigational for this application

doi: 10.1016/j.jdermsci.2009.11.005

Neurotoxicity was almost completely prevented by the presence of either NMDA receptor antagonists, MK-801 and D(-)-2-amino-5-phosphonopentanoic acid (D-AP5), or mGluR antagonist, (S)-4-carboxyphenylglycine (S-4CPG)

(PubMed) Hynes KL, Otahal P, Hay I, Burgess JR

Unlike Amino Asylum (raided) or Paradigm Peptides (charged), Peptide Sciences appears to have shut down proactively likely in response to increasing regulatory pressure rather than active enforcement

More precisely, an excessive amount of miR-9 causes a decrease in both the mRNA and protein levels of GOT-1, consequently diminishing the intracellular Fe2 + accumulation induced by glutamate and sulfasalazine [74]