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Moreover, the hyperhomocysteinemia associated with an increased cardiovascular risk is stronger in non-insulin-dependent diabetes mellitus than in normal subjects (Hoogeveen et al, 1998)

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There is also a lack of uniform specificity among pharmacological tools to modulate ferroptosis and there are many ways in which they can affect inflammation and metabolism independently of ferroptosis, thus requiring both genetic and lipidomic validation for strong inference

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10.1097/01.brs.0000096943.27853.bc 97 GruberH