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Lets take a closer look at the biological mechanisms that make this possible

Widely-accepted preventive strategies for CIN include volume expansion with intravenous saline or sodium bicarbonate and the use of low-osmolar or iso-osmolar contrast media [176]

It significantly ameliorates high-fat diet-induced structural dysbiosis of the gut microbiota by reducing the abundance of harmful bacteria positively correlated with hepatic lipid deposition (e.g., Lachnospiraceae and Collinsella) and increasing the abundance of beneficial bacteria (e.g., Ruminococcus and Turicibacter), thereby reducing hepatic lipogenesis and ultimately alleviates liver steatosis and lipid accumulation ( 4.4 Autoimmune hepatitis (AIH) Autoimmune hepatitis (AIH) is a type of liver inflammation characterized by the immune system attacking hepatocytes, leading to chronic liver injury ( The concanavalin A (ConA)-induced liver injury model is commonly used to study AIH

These are guidelines, not prescriptions

Cell Rep 10 , 17221734 (2015)

An additional insult or pathogenic factor, often referred to in the literature as the second hit, is required to trigger the progression from simple steatosis to steatohepatitis (MASH), characterized by hepatocellular injury, inflammation, and varying degrees of fibrosis [32]