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Some studies propose that macrophage factor IL-1 and Th17 cytokine IL-17A may negatively impact GR function by upregulating the expression of GR subtypes, with GR being the primary structure exerting effects (335, 336)

critical information on tissue penetration, metabolic half-lives, and clearance pathways is largely absent from the literature, undermining the ability to predict therapeutic windows or potential toxicities

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