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This observation presents a potential parallel to findings in cancer research, where DNA methylation directly governs GPX4 expression (218)

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In: Shively J.E, editor

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However, the in vitro data indicate that this cytotoxicity and the perforin expression levels in NK cells appear to be dependent upon endogenous IGF-1 and miR-483-3p expression, given that they can be inhibited by either IGF-1-neutralizing antibody or miR-483-3p overexpression