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doi: 10.3389/fimmu.2021.719023 132 KalraR

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mTOR and ATF4 constitute a core regulatory axis that often operates through a positive feedback loop to amplify amino acid metabolic flux, mTORC1 signaling can enhance ATF4 expression [36]

Tocai, A

Cells use glutathione to manage oxidative stress, support detoxification pathways, and keep the balance between damage and repair inside the cell

As seen above, key molecular targets include Beclin-1, which regulates autophagosome formation, and NHE1, whose inhibition prevents maladaptive autophagic cell death