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The neuroprotective treatment of AIS mainly targets ischemic cascades, such as mitochondrial dysfunction, oxidative stress injury, neuroinflammation, and programmed cell death processes including apoptosis, pyroptosis, and ferroptosis

Centrally acting neurotransmitters including noradrenaline and serotonin are believed to be involved

Accessed 28 Feb 2020

Histology showed 2.5-fold fibroblast proliferation
