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This in turn induces dysfunction of tumor-specific CD8 + T cells and contributes to adaptive resistance to immunotherapies [126,127,128,129,130]

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Following primers were used to generate the W114A mutation: forward 5-GCTCTTTGAATTCGGGCTACCGAGTAG-3 and reverse 5-CTACTCGGTAGCCCGAATTCAAAGAGC-3, as well as forward 5-GCTCTTTGAATGCGGGCTACCGAGTAG-3 and reverse 5-CTACTCGGTAGCCCGCATTCAAAGAGC-3