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Iron metabolism plays a central role, as excessive intracellular iron promotes the Fenton reaction, generating lethal amounts of ROS that drive lipid peroxidation [5]

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visual acuity increase, extension of visual field borders, and normalization of electrophysiological indices) in more than 90% of cases

Furthermore, a membranome-based approach on immortalized NP and AF cell lines established from adolescent healthy disc tissue yielded various membrane-associated markers in the AF and NP (van den Akker et al., 2020)

Research shows that SFN can restore NQO1 enzyme activity in leukemia cells carrying this polymorphism, which normally results in reduced activity ( Crucially, for the most extensively studied GST polymorphisms, the metabolic impact is profound and counterintuitive